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The Portrait of: Mr. Augusto S. Moaio The Mu-man “The Mu-men, how did they get here?” asked Professor Eceptico-Espirtu, of the University of Lima (in Peru). “How do you think,” said a youthful student named: Augusto S. Moaio, a wild looking flat faced undergraduate from one of the South Pacific Islands: adding, “they came on a damn ship from Saturn and some from Mercury, from its gigantic volcano area.” It was the first day of classes for the students and so the Professor hesitated in correcting the young lad, and simply smiled reluctantly at him. Then after a—something shorter than a pause—he remarked, “That all seems a bit far fetched, like one of those Edgar Rice Burroughs novels, or Mr. Doyle’s “Lost World,” crap”; the class laughed and so the professor figured he’d string the new student along and listen like a good father would to a spoiled son, and then make a lesson out of him in front of the class. “So it does,” responded the mad and impatient young man, with a receding hairline, and long ears; not long-long ears, but not normal size either. Matter of fact, the professor took a second look for he had not noticed them a moment ago being long at all. Said the Professor [cynically] “Tell me Mr. Moaio just where these Mu-men came from in a more specific and detailed manner: and if possible, in chronological order, for we all seem so uninformed according to you; henceforward son, move on, give us a better grasp on this!” This was the normally way for the professor to scare off his challengers [or challenges] in class; that is, toss a little fun their way [belittle them if need be] make them sweat; thus, shutting down their stupid questions, or remarks, as he felt they were just annoyances, but he had to allow some inquiry. Said Mr. Moaio with a smile [after a short consideration], or was it a sneer, it’s hard to determine, “They were already here long before the aliens arrived: the Mu-men that is.” “You don’t need to clarify who we are discussing; you are all alive and I dare say, some undergraduates, and some graduate students, are you not; you all got cultured brains I hope, especially being in my class you better have.” The ‘not’ and ‘you’ had an inflection to it. “Carry on Augusto,” bellowed the professor. [A little stiffly—he’s mad.] “As I was about to say,” the class all looking at the young tall man standing by his desk now, all twenty students with inquisitive eyes and wondering if this was a stage play or what. “…the primitive Mu-men were injected with a chromosome buster, they were evidently breaking and life expectancy was less than twenty-five years for them, and the aliens helped in this area, in particular, the Saturnites. This of course was the beginnings of the highbred Mu-men, whom were similar to our great apes or primates if you will, prior to their helpful technology.” The professor now said [laconically]. “So are we getting a lesson on Evolution, Mr. Moaio?” “Oh no just a chronological order of how they came to be and whom they were as you wanted Sir.” “Carry on, carry on, young lad…” said the professor—wild-eyed—with distain in his countenance, adding: “and when did all this take place, since you seem to have hidden knowledge none of us have; dates give me dates, they got to someplace in that big head of yours.” Now the professor got another laugh from his students, as he predicted. But it didn’t seem to faze the new student. “Well,” he said with thought through breathe, ‘it’s not all that simple, it really was a long trip, I mean it happened in stages….” [A pause, as Augusto took a swallow.] (The professor now leaned against his podium, putting his forearms down on its wooden side frame; his lecture was stopped for the most part and he knew it, which was originally on the 8th continent [Lemuria: which was to have stretched from Easter Island to Tahiti, to Fiji and onto Guam and beyond, and over to Hawaii]. He was going to explore the Maya culture and the Egyptian and try to mix it in with Lemuria. It was all lost now, the South Pacific per se was his domain to talk about, he had spent 26-years on Easter Island, during his summer breaks, and was always delighted to start his program out on the history of this area adding his exploits to the learning process, and this Augusto had just taken it away.) Said the professor [emphatically], “Were you were about to say something Mr. Moaio?” [Blinking.] “The Mu-men were once a great ape society, giants if you will (the professor quickly added, ‘Like King Kong I suppose?’ but Augusto just continued to talk without stopping). In consequence, they were given a Gravity-reinforcer, what you might call a membrane around a cell, but it was put around the chromosomes of the Mu-men, allowing their chromosomes to withstand their breakage so easily. And in time they were even given an additional chromosome. Again I repeat myself, allowing longer life for the Mu-men. The collapsing of the chromosomes was the big fault the aliens from Mercury had concluded. Thereafter, their life span jumped up fifteen if not twenty-five years, and as time proceeded they would gain even a longer life span, once acquiring better eating habits, disease control, along with better hygiene. I do agree with you professor with the size of the continent, although it was a bit larger (the professor gave a limped smile). The Mu-men were self producing, in essence, they kind of laid eggs in reproducing themselves. And by the continued aid from the two alien races, they acquired both sex organs, and started to cohabitate with humans. Actually capturing them and bringing them to their abodes as they felt a need to, or out of necessity for offspring that might be more humanoid like. As a result, the alien races decided to stop the so called experiment; of course to the disappointment of the Mu-men. Let me add, the Mu-men were now a distorted bunch of creatures: some with three eyes, and feet that looked like ducks so they could walk backwards or forwards, some even sideways. In addition, they had a small cranial, possible that of the Neanderthal, or even Homo erectus. But he or they did become a new species, and that was what they wanted.” As Augusto stopped to catch his breathe, the professor noticed his brow ridges were pronounced over his eyes (he hadn’t noticed them before being so), it was as if he was of an old age; for he concluded, age, thickens the brows, and drops the jaw bone, thus he must be very old, but he was young looking in all fairness. The Professor [losing confidence under Augusto’s stare] said, “Continue please,” digging his fingers into the wood of the podium stand. [Cooley.] “Well,” he continued with a dry mouth, but steady voice, “they had little brains compared to us, one could say. But great was their supernatural willpower; that is to say, they could move objects unbelievable heavy. Things large cranes today could not move.” [Suspiciously.] The professor looked up to the ceiling as if to stop Augusto from talking for a moment—showing a bit of world-weariness, and want to insert his two-cents worth, thus, saying as he lowered his head, “No, no, now do you really think we are to believe this, I mean, move what, show us, I mean point to an example so we can scientifically …” [Augusto now interrupts. He rings off despondently.] “I was about to explain, if you will let me Professor [a pause, limited to a moment] the Mu-men moved great stones with the clap of an eye, how they acquired this ability was a mixture of their hybrid genetic breeding I would imagine. They were quite primitive you know, and had four arms at one time. And for your dates, I’d say it was 17,694 BC when they became completely a jawboned bipedal human, yet let me not forget to include for your information, they remained still linked to the ancestry of the two limbed Lotus Demon [of Mercury] now, they carried their blood through these developing stages of trying to become closer to the humanoid species. And then around 13,500 BC, the war started with Atlantis.” “Honesty,” said the professor, “my gosh, now we got Atlantis in this so called thesis, and a two limbed demon, what next?” Two limbed Lotus Demon Said the professor with a speculative eye, “It seems to me you are grabbing at fragments of unwritten, mythological history, legends if you will, adding them to your recipe of anthropological gobbledygook, and with a slice of interplanetary jargon; and thinking we are to swallow it whole?” Augusto (with a tortured mind trying to convince the professor ((magnanimously))—assured himself he’d give it one more try), “Professor [he said], a large object, possible several miles across struck the planet Mercury, this smashing into the planet caused immense waves of superheated vapor that rolled for hundreds of miles, killing everything in its path, thus the Mercurynites sought out another haven, earth. The impact was so devastating it caused a tidal wave sending millions of tons of dust and vapor into its atmosphere, which darkened a side of the planet; in a similar manner the very thing that took place on earth. The creatures of Mercury are in our blood.” Augusto had to imply the word ‘us,’ instead of ‘him,’ so as to not cause alarm. Mercury’s Demise At that very moment Augusto sat down in his chair, closed his eyes, and folded his hands [somewhat despairingly]. The Professor noticed now he had long finger nails—so the professor had just noticed—with a lofty high head of red hair, again something that just occurred to him, and his groin area bulged out as if he had an overgrown penis. All concerned, he was looking [He being: the Professor] at the rest of the class to see if they had noticed the transformation of this young student’s bodily configuration—and to no avail, they all seemed quite content to carry on with listening to the dialogue between the two, without an iota of any x-ray appearances taking place. Thus, he rubbed his eyes and wiped his glasses, but it was more than that. He tried to place this person into a gap of time, pre-historic epoch, relating him to mankind’s ancestors, like: Australopithecus, Homo Habilis or Home erectus, for he was shape changing in x-ray vision in front of him with such features, yet his height remained the same. Possibly he was seeing layers of this person, his ancestry layers, along with bazaar alien layers also, such as: skull, lower jaw, ribs, and vertebrae and limb fragments, ex-ray configurations. He was no paleontologist, but he knew what he saw in the fossil findings of early man, and he knew anatomy quite well. And he concluded he was witnessing 40,000-years in a moment’s time. As Augusto closed his eyes, he held his hands against his frontal lobe, he chanted something beyond recognition, the professor could hear his heart beat, it was like the thumping of hoof beats—hoof beats coming louder and louder; the professor became speechless, almost as if in a trance. To break the silence the professor said, “It is all still a mystery; just, just a damn mystery…” but at the end of the last word the five story building started to shift off its concrete foundation, brick by brick it loosened and lift its home base—lifted up several inches from its groundwork. Then the young man opened his eyes, a flat look on his face, his teeth grinding, eyes bloodshot like a gorilla’s, a Great Ape’s. Said the young man with a tarnished and rustic voice, one not quite like the Professor had heard a few minutes ago: “Mysteries are not meant to be completely sold for the price of curiosity, they all have a heavier price than one normally wants to pay, and should you wish to seek out all it has to offer, you will have to pay the price.” It was a statement not a question. It was as if the lad was giving the professor a choice of some kind (we also must remember the building is still standing several inches in the air and throughout the hallways and classrooms people are thinking an earthquake just took place and are running wildly about.) But let me continue with the shrewd professor—so he thinks he is. “Mysteries, the mind, the why’s, they belong to people like me, who have studied all their life to seek them out; the layman knows not how to handle such things, it is the scientist who deserves the discovery.” The young man just looked [eagerly] at the professor as if he may get his wish. Then [breathlessly] crashing through the door was the Dean, he had ran from classroom to classroom, but when he came upon Professor EE’s room [as he was often called] he was stunned to see everyone still sitting calmly, and the professor at the podium still having a discussion, or so it looked like it to him. “Are you mad Professor EE, get this classroom out of harms way, get them outside, we’re in the middle of an earthquake!” then he ran uncontrollably out of the room to warn the adjacent class. At that moment, that very moment, the class seemed to have gotten out of its fog and stumbled to the door, all left, but the professor and the young man, whom remained stationary in the same positions they had been for the past hour, with their ongoing dialogue. “Ah!” said Augusto [fiercely], “there is a Mecca of possibilities Professor!” The professor knew beyond a doubt he was with some kind of ancient being; possible a shape-changer, things were too weird, the whole day was too eerie. The building now fell back roughly onto its foundation, but was still not stable, it was leaning, and some of floors and stairways had broken and sunk onto the lower floor; it would take a miracle to put it back into place; it would have to be rebuilt. The Professor [astounded] asked, “Where are you from?” now having changed his style and tone of voice. “From the third cataclysm of Atlantis and the one wherein Mu sank, and Atlantis survived; as it had twice before tasted near-extinction, calamities as you would have it. The forth cataclysm it sank completely, those who survived, were scattered around the world. The residue of Mu was scattered around the world likewise, I helped build the Gran Saposoa in the Amazon jungle, lost to humanity for 2000-years. I seen two Ice Ages come and go; I witnessed the warm airs of Europe pass over to North America when there was no Greenland to subdue it. I witnessed the Geological North Pole move from the Northwest Passage to where it is today. I was one of the first Chahopoyas natives. It’s been an interesting life to say the least.” A sneer again appeared on the professor’s face, Augusto knew he’d have to prove it, but should he it would have to be—aggravatingly. It was one thing to show his powers in levitation, another to say you were over 13,000-years old. “Excuse me Professor,” said Augusto, “just how much proof do you want of me, to scornfully prove, the Mu-man lives on in me?” Now Augusto’s body became like an x-ray again, but with beams radiating from it. But the professor, arrogantly would not except this manifestation as proof he was as old as he claimed or personified in [with] his materializations; and Augusto could not go beyond this without harming himself, or for that matter, without returning to his old genetic half-human like species, the one he left behind so long ago; changeability was not on his menu like his grandfather’s before him: it would be his obliteration, he had chromosomes now that would never break, he could live possibly 20,000-years should he care for himself properly. (You could hear the fire engines, and the police cars now outside ((below)); the authorities wondering what had, and was taking place, while these two men remained standing in the same place, same position they had now for, let’s say an hour and a half. Then just as the professor began to laugh, a little stiffly he became, his bones were receptive to the new developments inside his skin; his chromosomes: his twenty-third lost its vitality—his face looked as it had gone mad, his chin drooping with old age, distorted; he was developing long lived hormones, he was separating from the Homo sapiens, more within the genera of Australopithecus, with features closer to the Neanderthal, thus he was becoming a living fossil, if you will: close to the looks of Homo erectus. His large brow ridges now rested over his eyes, made him look a thousand years old, a build-up of bone over the eye socks that were so pronounced he could not look straight up at the ceiling as he did before; his feet were like a ducks, he must had been nine feet tall now, with a three eyes, two new arms growing, facial distortions, worse than homo erectus; a primitive human species beyond his imagination, more like the Murcerynites. His brain capacity was lowered, he couldn’t think quickly, and when he did think and try to hold the thought, he forgot it even quicker, but he had a stronger will now, but didn’t know how to use it. He would soon find out, he couldn’t change his body back to how it was. Augusto had learned how to transform into another comatose body, and when that person died of old age, he’d shift into another. But this freak of nature, as the professor would soon be, would be subject to all the sciences the world had to offer. He would never have peace. That is when Augusto stood up, walked out of his the classroom, never to return; for the shrewd professor could not speak a language anymore, just some sounds, gestures, and he became the talk of the decade, until he committed suicide. penis elargement surgery cost penile enlargment review do penis enlarement pills really work vimax penis enlargement product best penis elargement surgery safe penis elargement penis enlarement excercises vimax penis enlargement pills review

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Medical hair restoration in the literal sense includes the hair loss treatment which depends upon the use of medicines. Unusual hair loss both in men and women is caused by the alterations in the androgen metabolism. Androgen is a male hormone which has a major role to play in regulation of hair growth or hair loss. The dermal papilla is the most important structure in a hair follicle which is responsible for hair-growth. It is the dermal papilla, the cell of which divides and differentiates to give rise to a new hair follicle. The dermal papilla is in direct contact with blood capillaries in the skin to derive the nutrients for the growing hair follicle. Research has shown that dermal papilla got many receptors for androgens and there are studies which have confirmed that males have more androgenic receptors in dermal papilla of their follicles as compared to females. The metabolism of androgen involves an enzyme called 5 alpha reductase which combines with the hormone androgen(testosterone) to form the DHT (Dihydro-testosterone). DHT is a natural metabolite of our body which is the root cause of hair loss. Proper nutrition is critical for the maintenance of the hair. When DHT gets into the hair follicles and roots (dermal papilla), it prevents necessary proteins, vitamins and minerals from providing nourishment needed to sustain life in the hairs of those follicles. Consequently, hair follicles are reproduced at a much slower rate. This shortens their growing stage (anagen phase) and or lengthens their resting stage (telogen phase) of the follicle. DHT also causes hair follicle to shrink and get progressively smaller and finer. This process is known as miniaturization and causes the hair to ultimately fall. DHT is responsible for 95% of hair loss. Some individuals both men and women are genetically pre-disposed to produce more DHT than the normal individuals. DHT also creates a wax-like substance around the hair roots. It is this accumulation of DHT inside the hair follicles and roots which is one of the primary causes of male and female pattern hair loss. Blocking the synthesis of DHT at molecular level forms the basis for the treatment of MPHL ( male pattern hair loss) and FPHL female pattern hair loss). There are many natural DHT blockers and a number of drugs which are used for medical hair restoration. Let us see the main drugs which are available for medical hair restoration in men and women. Minoxidil Minoxidil has the distinction of the first drug being used for promoting the hair restoration. This medical hair restoration treatment drug was used earlier as an oral antihypertensive drug, but after its hypertrichosis (excessive body hair) effects were noticed, a topical solution of the drug was tested for its hair growing potential. Minoxidil was then approved as medical hair restoration treatment drug for men by the US Food and Drug Administration (FDA) in 1988 as a 2% solution, followed by 5% solution in 1997. For women, the 2% solution was approved in 1991. Though 5% solution is not approved for women, it is used as a medical hair restoration treatment by many dermatologists worldwide. Both solutions are available without a prescription in the US. Mechanism of action Minoxidil is thought to have a direct mitogenic effect on epidermal cells, as has been observed both in vitro in vivo. Though the mechanism of its action for causing cell proliferation is not very clear, minoxidil is thought to prevent intracellular calcium entry. Calcium normally enhances epidermal growth factors to inhibit hair growth, and Minoxidil by getting converted to minoxidil sulfate acts as a potassium channel agonist and enhances potassium ion permeability to prevent calcium ions from entering into cells. Thought the exact action of minoxidil preventing the formation of DHT has not been shown but the drug has been shown to have a stabilizing effect on the hair loss. The result of the drug takes about few months time to be evident since it is the time which is necessary for restoring the normal growth cycle of hair fibers. Use of Minoxidil has approved by FDA for men (Norwood II-V) and women (Ludwig I-II ) older than 18 years. It is used as a medical hair restoration treatment either for frontal or vertex scalp thinning. It brings about an increase in density which is mostly caused by conversion of miniaturized hairs into terminal hairs rather than a stimulated de novo re-growth. The hair loss becomes stabilized after continued use of drug, which takes about a year’s time for the medical hair restoration treatment to show its complete results. Hair loss restoration treatment with 0.05% betamethasone dipropionate and 5% topical minoxidil are found to be superior to minoxidil alone. Topical minoxidil is very well tolerated and adverse effects are mainly dermatologic. The most frequent adverse effect is an irritant contact dermatitis. Though minnoxidil does not have any effect on blood pressure, it should be used with caution in patient with cardiovascular diseases. It is also contraindicated in pregnant and nursing mothers. Finasteride The drug finasteride was earlier used as treatment for prostate enlargement, under the medical name Proscar. But in 1998, it was approved by FDA for the Medical hair loss restoration in MPHL. Mechanism of Action Medical hair restoration treatments with Finasteride depends upon its specific action as an inhibitor of type II 5α-reductase, the intracellular enzyme that converts male hormone androgen into DHT (Dihydro Testosterone). Its action results in significant decrease in serum and tissue DHT levels in even in concentration as low as 0.2mg. Finnasteride is able to stabilize hair loss in 80% of patient with Vertex hair loss and in 70% of patients with frontal hair loss. Most of these patients are able to grow more hair or retain the ones they have. The peculiar thing about Propecia is that its effect is more pronounced in crown area than in the front. The hair that grow after the medical hair restoration treatments are better in texture and are thicker, more like the terminal hair. The best thing about medical hair restoration treatment with the finnasteride is that it is well tolerated and has minimal side effects. Sexual dysfunction (decreased sex drive, erectile dysfunction, and decreased semen volume) are observed in about 3.8% of cases. But these side-effects subside within few months of Medical hair restoration treatments or disappear within a week’s time as soon as the treatment is stopped. It generally requires about 6 to 12 months for the m edical hair restoration treatment to be apparent but the side effects appear earlier. So even after the medicine is stopped, there is no possibility of loosing the hair that has been gained, but the side effects are sure to disappear. Many hair restoration surgeons find Propecia (finasteride) to act as an excellent adjunct to the surgical hair restoration. There are several benefits of this kind of combination therapy. As the Medical hair restoration with Propecia brings about a hair re-growth in the crown area, it has a complementary action; it allows the surgeon to have more donor hair to be available for frontal hair transplant and design the hairline at his own will. Since finasteride has no effect in the frontal area of the scalp, it does not have any interference with the surgical hair restoration. Combination Therapy There are reports which say that use of finasteride and topical minoxidil combination therapy as a Medical hair restoration treatment is of more advantage in cases of mild to moderate MPHL. Further studies are in progress. Many hair restoration doctors have already started the use of combination therapy in order to obtain better hair growth. Anti Androgen Therapy For women with hyperandrogonism( with increased levels of androgen) who do not respond well to minoxidil, antiandrogen therapy is another option of Medical hair restoration. In UK the most commonly used anti-androgen for women is CPA (cyproterone acetate), which is used in combination with ethinyl-estradiol. However, in United States, where CPA is not available, the aldosterone antagonist spironolactone is the alternative choice of hair restoration doctors. Flutamide Medical hair restoration with flutamide has shown improvement as hair loss restoration treatment in women with hirsutism. For hyperandrogenic premenopausal women, flutamide is a better medical hair restoration agent than both the CPA or finestride. Hair loss restoration management is a structured process which depends upon many factors along with the medical hair restoration. For more details on the topic you can refer to section medical hair restoration or article on male pattern hair loss or female pattern hair loss at our site hairtransplantadvice.com. does penis enhancement work penile enlargement pills product herbal penis enhancement vigrx review vimax penis enlargement information penile enlargement pills penis enhancement video prosolution pennis enlargement pills free penis enlargement pills

Almost all human beings are conscious about their beauty and they adopt all means to improve or maintain it. The quality of skin is having priority in the field of beauty care. Skin is considered as the mirror of the human body and it reflects the physical, mental and emotional status of every person. The number of people attending beauty clinics and dermatologists is increasing day by day. Even though skin diseases are having low mortality, people are worried about skin problems because of the social stigma. A person having a disease of some internal organ may not be worried but another person having a skin disease on the face may be depressed because of the simple reason 'others will come to know about my disease'. Human skin has got several functions like protection, heat regulation, water balance, excretion etc. It gives protection and support to the deep body tissues and has got the ability to stretch to some extent due to the presence of protein fibers in the dermis. But too fast enlargement of the body parts cause excessive stretching on the skin resulting in breaking of collagen and elastin fibers of the dermis. This causes slightly depressed discoloration called stria atrophia or stretch marks. Initially these marks are slightly pinkish and later become white or silvery in appearance. Stretch marks are found in conditions like pregnancy, puberty, obesity etc. It is usually seen in places like abdomen, thighs, breasts, buttocks, shoulders etc. Once these marks appear, time is required for them to be less prominent. Both men and women are the victims of stretch marks but it mainly affects women due to their beauty consciousness and causes psychological trauma leading to depression. On the other hand there are women who are least bothered to get rid of stretch marks and consider them as a badge of mother hood. Still the number of people visiting clinics to remove stretch marks is increasing day by day. Because of the increasing need for the stretch mark treatment modalities, there are number of products in the market to deal with stretch marks cure. But most of those products are not proved effective scientifically but few of them give results in many cases. Modern treatment methods like plastic surgery and laser surgery for stretch marks have been proved effective in the treatment. prosolution pnis enlargement pills penis enlargment tool best penis enlargment surgery penile enlargment testimonials penis enlagement surgeon free penis enlargment tip penile enlargment excercises pennis enlargement surgery picture free penis enlargement pills

If you’ve ever caught someone in the act, then you’ve certainly felt a little warmer in that moment. You thought about being there, and feeling what they were feeling. Just talking about it makes me all excited. So when you’re swinging, what about looking at the scenery? Well, that certainly has its place as well. Opening Your Eyes When you’re first in a swinging situation, you may feel almost too aroused to do anything but watch. For the first few times I was with my partner, we just looked at the other couple in awe. For one, we were in awe of their openness. And for two, we were excited to see someone else being pleasured. Just when you think you’re not turned on by something, then you try it and realize, wow, that’s amazing. I think that many people feel ashamed to say that they like to watch others have sex or touch each other. And why should we? It’s a completely natural act. And how empowering is it to be absolutely comfortable to watch someone else? Very. Will The Other Couple Mind? If it’s just you two and another couple, then you may want to broach the subject ahead of time. And if they say that it’s okay, then you can feel completely comfortable to watch them in action. You may also feel aroused and follow suit, or pleasure yourself as you watch. Again, clear this with them ahead of time. Most couples won’t mind if you ask. And if you’re in a swingers club, it’s almost guaranteed that you’re going to be seen if you start anything, so it’s a free for all for voyeurs. Remembering to blink may be hard though. What If We’re Asked To Perform? Think of being the subject of a fantasy as being in your favourite erotic movie and you are the stars. Forget about who is watching and let the moment take you and your partner to ecstasy. Pleasure him or her as you normally would. Touch their skin, feel their thighs, slowly tease the sighs out. If you really want to make an impression, open your eyes and look to the other couple. Let them know how much this turns you on, and how wonderful something feels. Why Do You Look? Looking at difference shapes of bodies is intriguing. And in a comfortable setting, you will see that it really isn’t the size that matters, but the confidence of the person. Women aren’t judged by their weight and men aren’t seen for the length of their penis. Men aren’t counting another man’s abs, and women aren’t considering the bounce of the breasts. You look because you’re curious, but you continue looking because you can see the beauty in everyone. In most cases, looking will only satisfy so much and you will want to be the ones looked at. And that is completely natural and enthralling. Knowing that someone is watching you and getting pleasure from it can turn an ordinary moment into an erotic scene. buy pnis enlargement pills natural penis enhancement prosolution penis enlargement pills natural penis enlarement technique vimax free penis enargement exercise free penis enlargement exercise free pnis enlargement technique free penis enlargement pills

Being a natural athlete I’ve always sought to find more ways to increase testosterone levels naturally. The question I’ve always asked myself was what are the variables, which determine how much testosterone is boiavailable. I’ve read articles before, giving some advice on what in my every day life causes my test levels to go up and what causes them to go down. But after an extensive research I couldn’t find an article, explaining in detail how and actually what exactly I should manipulate directly in my system to achieve the effects I desired – namely not only higher levels of circulating testosterone but also how much of it will be available to the corresponding receptors in the cell walls. Moreover, I wanted to know all this in regards to natural bodybuilding… So, I decided to do some good digging and reading and then if I manage to put things together, to lay that in writing form so that others can benefit as well. Without wasting even a minute more I will start this off by describing what testosterone is and what it does in the human body. Testosterone is a steroid hormone with anabolic and androgenic properties. It is the main hormone, responsible for the increase in lean muscle tissue, increased libido, energy, bone formation, and immune function. Testosterone is secreted in the testes in men and in the ovaries in women. Small amounts are also secreted in the adrenal glands. Testosterone is derived from cholesterol. The levels of testosterone in men range between 350 and 1,000 nanograms per deciliter (ng/dl). After the age of 40 these normal levels start dropping by roughly 1 per cent a year. In the blood stream testosterone circulates in great percentage bound to so called binding proteins. Sex Hormone Binding Globulin or SHBG is the one that concerns us the greatest. Why? Because this is the main reason why testosterone might not be available to reach the cell receptor. When testosterone gets attached to SHBG, it is no longer able to perform its anabolic functions. What elevates SHGB: Anorexia nervosa, Hyperthyroidism, Hypogonadism (males), Androgen insensitivity/deficiency, Alcoholic hepatic cirrhosis (males), Primary biliary cirrhosis (females). What suppresses SHGB: Obesity, Hypothyroidism, Hirsutism (females), Acne vulgaris, Polycystic ovarian disease, Acromegaly, Androgen-secreting ovarian tumors Less than 1% of the circulating testosterone is in a free form in males (less that 3% in females). Only when in a free form this hormone can exhibit its properties by connecting to the androgen receptors on the cell walls. Based on a study 14 to 50 per cent of the testosterone is bound to SHBG in males and 37 to 75 in females. It is worth mentioning that SHGB poses very high affinity for binding to testosterone. Therefore, changes in the SHGB levels noticeably influence the level of bioavailable testosterone. Let’s discuss for a moment what exactly a testosterone bioavailability is. Other than SHGB there are two more testosterone-binding proteins, also called carriers. One of them is albumin. It is a low-affinity binding protein, thus testosterone bound to it is considered “bioavailable”. Albumin binds to testosterone in the range 45 to 85 per cent in men (25 – 65 in women). The third carrier is the cortisol binding globulin, which binds also with low-affinity to less that 1 % of the testosterone in circulation. The free androgen index (FAI) indicates the amount of bioavailable testosterone. FAI is the sum of the free testosterone and the albumin and cortisol binding globulin. Or it’s the total serum testosterone minus the SHGB-bound testosterone. It is now clear why we should focus our attention on the properties of SHGB. The levels of this binding protein increase when there is excess estrogen present. Conversely, SHGB levels drop if the testosterone levels are elevated. Here I should mention the fact that SHGB exhibits higher affinity to testosterone than to estrogen. Now, pay close attention… It’s a well-known fact that testosterone is an estrogen precursor – it will convert to estrogen under the influence of the enzyme aromatase. Nothing that we don’t know so far. Here is where it gets interesting. Suppose that we have normal testosterone levels and we don’t suffer from any of the health ailments, which influence the SHGB levels. That means that SHGB levels are normal, too. Bare with me now. If more of this testosterone is converted to estrogen due to abnormal aromatase levels, the SHGB I will increase as well. SHGB, being more readily bound to testosterone, will leave us with excess estrogen levels in the system, which in turn will stimulate increased production of the SHGB protein from the liver. This whole process ultimately amplifies estrogen levels. Estrogen readily binds to the androgen receptors in cells thus leaving less opportunity for the free testosterone. Even more important, estrogen is the messenger molecule that signals the brain to decrease testosterone production. Another thing of great importance is the fact that over 40 per cent of the SHGB protein circulates unbound in the blood stream in man (over 80 per cent in women), and albumin circulates unbound almost all of the time. Thus increase in the total testosterone levels does not produce any noticeable changes in the free testosterone levels unless there is a significant increase like the one seen after synthetic steroid hormone administration. Well, this whole story brings us to the conclusion that the main approach should be - to keep the testosterone bioavailability high. In this regard a natural athlete should strive to: · Prevent testosterone levels from getting low · Attempt to increase the total testosterone as a means of keeping the testosterone levels from plunging · Block the testosterone-binding effects of SHBG · Lower the levels of the enzyme aromatase - less testosterone conversion to estrogen 1. How to prevent testosterone levels from getting low? - Obesity. Based on the way the testosterone-estrogen mechanism works, increased levels of estrogen will ultimately decrease the circulating testosterone. Excess fat causes more estrogen production due to the fact that fat cells are those, which manufacture estrogen. So, the more fat cells, the more estrogen in the blood and the less testosterone. - Drug and alcohol abuse. Alcohol has the property to inhibit your ability to remove estrogen from the blood stream by acting as a central nervous system depressant and also by decreasing zinc levels. - Stress elevates corticosteroid levels in the blood steam, which causes the testosterone levels to decrease. - Medications. Some medications, including estrogen and progesterone, lower the lutenizing hormone (LH) levels. LH is the hormone, responsible for the steroid hormones production. - Diabetes. Studies suggest that there is a link between type 2 diabetes and lower testosterone levels. - Hypertension and high cholesterol levels. These both cause the arteries to harden, this way decreasing the blood flow to the sex hormone producing organs. That of course leads to low sex hormone levels. - Aging. Fact is after the age of 40 test levels drops by roughly one per cent per year. We can’t do too much about that. The clock keeps ticking for all of us. However, we can try to manipulate testosterone levels in any other possible way. - Low fat diet. Low fat consumption causes increase of the SHGB, which means one thing – less free testosterone. It is considered that monounsaturated fats play an important role in testosterone levels and bioavailability. - Overtraining can contribute to as much as 40 per cent drop in testosterone levels. That is why it’s important to notice early the signs of overtraining and give the body a week or two well-deserved rest. - Not enough sleep. If you are not getting enough sleep the body is not recuperating well, which causes less testosterone and more corticosteroids to be released. Just to mention corticosteroids like cortisol are in fact catabolic hormones meaning they use up muscle tissue to provide the brain and the heart with energy. - Vitamin C. It suppresses the release of the stress hormone cortisol. Cortisol decreases testosterone levels. So, ultimately less cortisol, more testosterone. 2. How to boost testosterone levels - Incorporate basic movements that involve several muscle groups in your training routine. Good ones are squats, dead lifts, and military presses. Basic (compound) exercises have been shown to play an important role in the testosterone levels. - The greatest workout related testosterone production occurs with the use of heavier weights and lower rep range. A study shows that the best is 85 per cent of your one-rep max. - Tribulus terestris is a natural supplement, which has been shown in some studies to have the ability to increase the leutenizing hormone (LH) levels. As we already mentioned above, one of the functions of LH is to stimulate testosterone production by the testes. 3. Block the testosterone-binding effects of SHBG - Nettle root as a highly concentrated extract has shown to be effective at binding to SHBG and therefore it permits more free testosterone to circulate the system. It also acts as a 5-alpha reductase inhibitor. This is the enzyme, responsible for the conversion of testosterone to dihydrotestosterone (DHT), a more potent form of the male sex hormone, which causes prostate enlargement and ultimately cancer. Methanolic extract of nettle can also cut down the SHBG levels, which is another form of elevating the free testosterone in the blood stream. - Pygeum (prunus africana) is another herb, known to block the testosterone-binding effects of SHBG. Nettle root and pygeum extracts also benefit the prostate gland as a preventive treatment for benign prostatic hyperplasia (BPH) both by blocking the 5-alpha reductase action. - Avena Sativa is an extract from the straw of oats. It has somewhat different properties. It works by freeing bound testosterone, which increases the free testosterone in circulation. 4. Lowering aromatize levels - Zinc. The mineral zinc inhibits the aromatase enzyme that converts testosterone into excess estrogen. The recommended dose for inhibiting aromatase is 80mg daily. However, be sure the combined zinc quantity of all the daily supplements you are taking does not go over this benchmark. - Chrysin is a bioflavonoid that has shown a potential as a natural aromatase inhibitor. Chrysin is poorly absorbed in the system. It is found that when taken along with piperine, chrysin exhibits a lot better absorption. Supplementation with chrysin and piperine together might bring good results in reducing aromatase levels. How does this all apply to the sport of natural bodybuilding Let’s assume you are an individual, who exercises regularly and who doesn’t consider taking exogenous steroid hormones. With other words you are a natural athlete or enthusiast, who desires to keep his natural testosterone levels in the range, favoring good health and lean muscle mass build up. Here is what you do: 1. If you are even slightly overweight consider staring immediately a diet and training routine, tailored toward fat loss and lean muscle retention. 2. Consider also dropping the alcohol intake to absolute minimum if you want your efforts in keeping test levels high to give results. 3. Begin mastering some types of self-control or even meditation. These will take care of the excess stress levels. 4. Take medications only if they are absolutely essential for your health and are prescribed by your doctor. Talk to your personal physician to find out if there are any natural remedies to replace your current medications and if he recommends such approach for your health issue. 5. Keep your blood pressure and the serum cholesterol in check. 6. Eat enough good fats in your diet. Good means monounsaturated and omega-3 and 6 polyunsaturated fats. 7. Don’t ever overtrain. Sleep enough to promote good recovery. Signs of overtraining are loss of appetite, tiredness and irritability, lack of motivation, impaired mental focus, prolonged recovery periods. 8. Start relying more heavily on basic exercise movements. Train in the low rep range most of the time. 5 – 8 reps will ensure that you’re using weights that will eventually stimulate elevated testosterone levels. 9. It will only do you good if you decide to try some or all of these natural supplements: chrysin - piperine blend, nettle root extract, pygeum, avena sativa extract, and tribulus terestris. Try to find them in your local health store. Follow the directions for best results. 10. And you shouldn’t even consider training without supplementing your diet with enough vitamin C (at least 1g a day) and zinc (15mg min.). Take vitamin C with your multivitamin formula after breakfast, and post-workout with your protein shake. Zinc should be present in your multivitamin blend but this is not enough. Take zinc with magnesium in the form of ZMA right before you go to bed on an empty stomach. References: Ron Geraci, Men's Health, December 25, 2000; 13 Ways to Naturally Boost Your Testosterone Levels LE Magazine January 2000; Replenish Testosterone Naturally Plant extracts favorably alter hormone metabolism and improve sexual desire in men Jennifer A. Kelly, Ph.D. and Leo Vankrieken, Eur. Eng. Diagnostic Products Corporation; Sex Hormone Binding Globulin and the Assessment of Androgen Status Cockatoo.com; Avena Sativa - are oats an aphrodisiac? Gabe Mirkin, M.D.; High Cholesterol Causes Low Testosterone Bodybuilding.com, Chrysin Info And Products - Block Estrogen, Increase Testosterone